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The effect of factors such as weight and other covariates on long term mortality outcome of tuberculosis patients in Texas. The study was reviewed and approved by the Institutional Review Boards (IRB) of UT Southwestern Medical Center (IRB# 122008020) and the Texas State Department of Human and Health Services: [(IRB #09-021) Principal Investigator: Ronald Hall].Definitions and classifications uti
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Ality in genitourinary tuberculosis was no different from latent tuberculosis, while meningitis had the poorest long-term outcome of 46.2 . Compared to meningitis the HR for death was 0.50 (CI: 0.27-0.91) for lymphatic disease, 0.42 (CI: 0.21-0.81) for bone/joint disease, and 0.59 (CI: 0.27-1.31) for peritonitis. The relationship between mortality and therapy duration for each type of EPTB was a u
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As then established for each patient. We compared allcause mortality rates between EPTB and of age-adjusted Texas population as well as of another comparable cohort of patients with latent tuberculosis treated during the same period [13,14]. The latent tuberculosis patient cohort was previously reported by us [13,14], while the Texas age-adjusted all-cause population mortality rates are published
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Ssion of the different S100 proteins is cell dependent and is regulated by specific growth factors, cytokines or activation of cell surface PRRs.225,227 Most S100 proteins are of rather small size (14 kDa) and are encoded by genes clustered on chromosome 1q21.228 One interesting exception to this rule is S100B, the coding gene of which is located on chromosome 21q22.3 and is found to be overexpres
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E wastebasket diagnosis, as "the other" which is not pulmonary. Methods: This is a retrospective cohort study of all patients treated for EPTB in the state of Texas between January 2000 and December 2005, who had no pulmonary disease. Clinical and epidemiological factors were abstracted from electronic records of the Report of Verified Case of Tuberculosis. The long-term outcome, which is death by
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R figure charges ?Immediate publication on acceptance ?Inclusion in PubMed, CAS, Scopus and Google Scholar ?Research which is freely available for redistributionSubmit your manuscript at www.biomedcentral.com/submitPusch et al. BMC Infectious Diseases 2014, 14:115 http://www.biomedcentral.com/1471-2334/14/RESEARCH ARTICLEOpen AccessTherapy duration and long-term outcomes in extra-pulmonary t
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Ssion of the different S100 proteins is cell dependent and is regulated by specific growth factors, cytokines or activation of cell surface PRRs.225,227 Most S100 proteins are of rather small size (14 kDa) and are encoded by genes clustered on chromosome 1q21.228 One interesting exception to this rule is S100B, the coding gene of which is located on chromosome 21q22.3 and is found to be overexpres
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